Type 3 diabetes and Metabolism with relevance to Genomic Medicine and the Global Diabetes Epidemic

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The current global diabetes epidemic in the developing world indicates that the incidence of diabetes is predicted to increase to 21% of the global population by 2050. In various continents the rise in the global diabetes epidemic has been associated with the pathogenetic involvement of various organ diseases such as non alcoholic fatty liver disease that is related to Type 2 diabetes, cardiovascular disease and neurodegenerative diseases such as Parkinson’s disease and Alzheimer’s disease. Therapeutics that involve genomic medicine may stabilize the severity of diabetes in various global communities and is required to prevent the early cellular senescence that is connected to the lifespan of Type 3 diabetic individuals. Skin changes are considered the most common manifestations of diabetes mellitus and interests in genomic medicine has accelerated to prevent skin barrier disorders before the onset of diabetes and to promote skin rejuvenation. Diet and liver disease are closely connected to various organ diseases and are of central importance with programmed cell death pathways. The central importance to nutrigenomics as early nutritional therapy may assist genes to delay skin, liver and brain disease associated with diabetes and aging. Anti-aging therapies such as diet, exercise and selective drug therapy early in life may maintain the activation of single gene that is important to molecular and genetic medicine and may control Type 3 diabetes that without genomic medicine accelerates skin diseases, cardiovascular disease, Type 2 diabetes and neurodegenerative disease. RELEVANT REFERENCS Martins IJ. Type 3 diabetes with links to NAFLD and Other Chronic Disease in the Western World. International Journal of Diabetes. 2016; 1:1-5. Martins IJ. Diet and Nutrition reverse Type 3 Diabetes and Accelerated Aging linked to Global chronic diseases. J Diab Res Ther. 2016; 2(2). Martins IJ. Magnesium Therapy Prevents Senescence with the Reversal of Diabetes and Alzheimer’s Disease. Health, 2016, 8, 694-710. Martins, I.J. Anti-Aging Genes Improve Appetite Regulation and Reverse Cell Senescence and Apoptosis in Global Populations. Advances in Aging Research, 2016, 5, 9-26 Martins IJ. Diabetes and Organ Dysfunction in the Developing and Developed. World Global Journal of Medical Research: F Diseases Volume 15 Issue 1 Version 1.0 Year 2015. CERTIFICATE OF PUBLICATION. Nutritional and Genotoxic Stress Contributes to Diabetes and Neurodegenerative Diseases such as Parkinson’s and Alzheimer’s Diseases. - Frontiers in Clinical Drug Research -CNS and Neurological Disorders. Volume 3 Editor: Atta-ur-Rahman Pp. 158-192 (35) DOI: ON LINE CHAPTER/EBOOK: 2015 Biography Dr. Ian James Martins is the Chief Editor of the International Journal of Diabetes Research and has joined the Editors of the Annals of Obesity and Disorders/International Journal of Diabetes Research and International Journal of Advances in Biology (2016). Reviewer for the Journal of Food Research/Annals of Neurodegenerative Disorders (2016) and previously for various other international journals. ResearchGate’s analysis available on www.google.com.au under Ian James Martins of Dr Martins’ publication stats place the SCIENTIFIC REPUTATION (RG score) higher than 96-97% of the international researchers with an H-index of 20 with 80 full text publications. Contribution: Peripheral sink amyloid beta clearance pathway with relevance to chronic disease and Alzheimer’s disease. Certificates of recognition at various international congress/conventions in various countries for invited speeches and keynote address (Ethnopharmacology 2016).