Abstract
In their interesting paper, Feder and co-workers (1996) propose an explanation for the genetic lesion in hemochromatosis. They implicate a sin gene some 4 megabases telomeric of the major histocompatibility complex (MHC) and offer the designation HLA-H based on similarities with HLA class I genes and neonatal Fc receptor (FcRn).
| Original language | English |
|---|---|
| Pages (from-to) | 336-340 |
| Journal | Immunogenetics |
| Volume | 45 |
| Publication status | Published - 1997 |