Abstract
In their interesting paper, Feder and co-workers (1996) propose an explanation for the genetic lesion in hemochromatosis. They implicate a sin gene some 4 megabases telomeric of the major histocompatibility complex (MHC) and offer the designation HLA-H based on similarities with HLA class I genes and neonatal Fc receptor (FcRn).
Original language | English |
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Pages (from-to) | 336-340 |
Journal | Immunogenetics |
Volume | 45 |
Publication status | Published - 1997 |