REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC

Research output: Contribution to conferenceAbstract

Abstract

Novel research indicates that the global non alcoholic fatty liver disease (NAFLD) epidemic is critical to the induction of the obesity epidemic. In man dietary and drug interventions in obesity have failed to correct the defect in the adipose tissue-liver interaction with the induction of NAFLD and NAFLD to effect between 20-40% of the global population. In various studies in obese animals dietary restriction has improved the adipose tissue-liver interaction with increased hepatic metabolism of lipids. Mitochondrial apoptosis is now a major concern in NAFLD with defective lipid metabolism in adipocytes related to adiposity and NAFLD. Specific genes have been identified that regulate the adipose tissue-liver interaction with calorie sensitive genes essential to maintain the interaction with the prevention of obesity. Global populations include various racial groups with consumption of anti-aging gene nutrients that may vary considerably between Caucasians, Africans, Europeans, Hispanics, Pacific Islanders and Asians. Nutritional and environmental epigenetics now identify the calorie sensitive gene Sirtuin 1 (Sirt 1) to be critical to the reversal of NAFLD and obesity. Sirt 1 is important to mitochondrial biogenesis with Sirt 1 repression responsible for the adipose tissue-liver defect. Nutritional diets that contain Sirt 1 activators reverse NAFLD with relevance to obesity and various global chronic diseases.

Original languageEnglish
Publication statusPublished - 18 Oct 2018
Event3rd International Conference & Expo on Obesity and Diet Management - Amsterdam , Netherlands
Duration: 18 Oct 201820 Oct 2018

Conference

Conference3rd International Conference & Expo on Obesity and Diet Management
CountryNetherlands
CityAmsterdam
Period18/10/1820/10/18

Fingerprint

Sirtuin 1
Obesity
Adipose Tissue
Liver
Lipid Metabolism
Genes
Essential Genes
Adiposity
Organelle Biogenesis
Non-alcoholic Fatty Liver Disease
Hispanic Americans
Adipocytes
Epigenomics
Population
Chronic Disease
Apoptosis
Diet
Food
Research
Pharmaceutical Preparations

Cite this

Martins, I. (2018). REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC. Abstract from 3rd International Conference & Expo on Obesity and Diet Management , Amsterdam , Netherlands.
Martins, Ian. / REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC. Abstract from 3rd International Conference & Expo on Obesity and Diet Management , Amsterdam , Netherlands.
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title = "REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC",
abstract = "Novel research indicates that the global non alcoholic fatty liver disease (NAFLD) epidemic is critical to the induction of the obesity epidemic. In man dietary and drug interventions in obesity have failed to correct the defect in the adipose tissue-liver interaction with the induction of NAFLD and NAFLD to effect between 20-40{\%} of the global population. In various studies in obese animals dietary restriction has improved the adipose tissue-liver interaction with increased hepatic metabolism of lipids. Mitochondrial apoptosis is now a major concern in NAFLD with defective lipid metabolism in adipocytes related to adiposity and NAFLD. Specific genes have been identified that regulate the adipose tissue-liver interaction with calorie sensitive genes essential to maintain the interaction with the prevention of obesity. Global populations include various racial groups with consumption of anti-aging gene nutrients that may vary considerably between Caucasians, Africans, Europeans, Hispanics, Pacific Islanders and Asians. Nutritional and environmental epigenetics now identify the calorie sensitive gene Sirtuin 1 (Sirt 1) to be critical to the reversal of NAFLD and obesity. Sirt 1 is important to mitochondrial biogenesis with Sirt 1 repression responsible for the adipose tissue-liver defect. Nutritional diets that contain Sirt 1 activators reverse NAFLD with relevance to obesity and various global chronic diseases.",
author = "Ian Martins",
year = "2018",
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note = "3rd International Conference & Expo on Obesity and Diet Management ; Conference date: 18-10-2018 Through 20-10-2018",

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Martins, I 2018, 'REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC' 3rd International Conference & Expo on Obesity and Diet Management , Amsterdam , Netherlands, 18/10/18 - 20/10/18, .

REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC. / Martins, Ian.

2018. Abstract from 3rd International Conference & Expo on Obesity and Diet Management , Amsterdam , Netherlands.

Research output: Contribution to conferenceAbstract

TY - CONF

T1 - REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC

AU - Martins, Ian

PY - 2018/10/18

Y1 - 2018/10/18

N2 - Novel research indicates that the global non alcoholic fatty liver disease (NAFLD) epidemic is critical to the induction of the obesity epidemic. In man dietary and drug interventions in obesity have failed to correct the defect in the adipose tissue-liver interaction with the induction of NAFLD and NAFLD to effect between 20-40% of the global population. In various studies in obese animals dietary restriction has improved the adipose tissue-liver interaction with increased hepatic metabolism of lipids. Mitochondrial apoptosis is now a major concern in NAFLD with defective lipid metabolism in adipocytes related to adiposity and NAFLD. Specific genes have been identified that regulate the adipose tissue-liver interaction with calorie sensitive genes essential to maintain the interaction with the prevention of obesity. Global populations include various racial groups with consumption of anti-aging gene nutrients that may vary considerably between Caucasians, Africans, Europeans, Hispanics, Pacific Islanders and Asians. Nutritional and environmental epigenetics now identify the calorie sensitive gene Sirtuin 1 (Sirt 1) to be critical to the reversal of NAFLD and obesity. Sirt 1 is important to mitochondrial biogenesis with Sirt 1 repression responsible for the adipose tissue-liver defect. Nutritional diets that contain Sirt 1 activators reverse NAFLD with relevance to obesity and various global chronic diseases.

AB - Novel research indicates that the global non alcoholic fatty liver disease (NAFLD) epidemic is critical to the induction of the obesity epidemic. In man dietary and drug interventions in obesity have failed to correct the defect in the adipose tissue-liver interaction with the induction of NAFLD and NAFLD to effect between 20-40% of the global population. In various studies in obese animals dietary restriction has improved the adipose tissue-liver interaction with increased hepatic metabolism of lipids. Mitochondrial apoptosis is now a major concern in NAFLD with defective lipid metabolism in adipocytes related to adiposity and NAFLD. Specific genes have been identified that regulate the adipose tissue-liver interaction with calorie sensitive genes essential to maintain the interaction with the prevention of obesity. Global populations include various racial groups with consumption of anti-aging gene nutrients that may vary considerably between Caucasians, Africans, Europeans, Hispanics, Pacific Islanders and Asians. Nutritional and environmental epigenetics now identify the calorie sensitive gene Sirtuin 1 (Sirt 1) to be critical to the reversal of NAFLD and obesity. Sirt 1 is important to mitochondrial biogenesis with Sirt 1 repression responsible for the adipose tissue-liver defect. Nutritional diets that contain Sirt 1 activators reverse NAFLD with relevance to obesity and various global chronic diseases.

M3 - Abstract

ER -

Martins I. REVERSAL OF NAFLD AND THE GLOBAL OBESITY EPIDEMIC. 2018. Abstract from 3rd International Conference & Expo on Obesity and Diet Management , Amsterdam , Netherlands.