Regulation of Body Temperature and NAFLD in Global Population

Research output: Contribution to specialist publicationArticle

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Abstract

https://www.asianhhm.com/.../regulation-of-body-temperature-and-nafld-in-global-po... The Non Alcoholic Fatty Liver Disease (NAFLD) epidemic is connected to diabetes, cardiovascular disease and neurodegenerative diseases. In the year 2050 it is expected that 30% of the world will progress to NAFLD. Recent scientific research indicates that the heat shock gene malfunctions in NAFLD with defective cell mitochondrial fat metabolism and heat production. Blood tests from blood biomarkers do not provide information for mitochondrial survival in NAFLD and chronic diseases with early death of cells in the liver, heart and brain connected to global chronic diseases.
Original languageEnglish
Pages1-3
Number of pages3
Volume1
Specialist publicationAsian Hospital and Healthcare Management
Publication statusPublished - 1 Feb 2018

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Body Temperature Regulation
Population
Chronic Disease
Thermogenesis
Hematologic Tests
Adipocytes
Neurodegenerative Diseases
Shock
Cell Death
Cardiovascular Diseases
Hot Temperature
Biomarkers
Non-alcoholic Fatty Liver Disease
Liver
Brain
Research
Genes

Cite this

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abstract = "https://www.asianhhm.com/.../regulation-of-body-temperature-and-nafld-in-global-po... The Non Alcoholic Fatty Liver Disease (NAFLD) epidemic is connected to diabetes, cardiovascular disease and neurodegenerative diseases. In the year 2050 it is expected that 30{\%} of the world will progress to NAFLD. Recent scientific research indicates that the heat shock gene malfunctions in NAFLD with defective cell mitochondrial fat metabolism and heat production. Blood tests from blood biomarkers do not provide information for mitochondrial survival in NAFLD and chronic diseases with early death of cells in the liver, heart and brain connected to global chronic diseases.",
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Regulation of Body Temperature and NAFLD in Global Population. / Martins, Ian.

In: Asian Hospital and Healthcare Management , Vol. 1, 01.02.2018, p. 1-3.

Research output: Contribution to specialist publicationArticle

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