Prolonged vitamin D intoxication: Presentation, pathogenesis and progress

Damon Bell; M.J. Crooke; N. Hay; Paul Glendenning

doi:10.1111/imj.12269

Prolonged vitamin D intoxication: Presentation, pathogenesis and progress

Damon Bell, M.J. Crooke, N. Hay, Paul Glendenning

Research output: Contribution to journal › Article › peer-review

17 Citations (Scopus)

Abstract

Vitamin D toxicity from unactivated vitamin D (calciferol) therapy is currently a rare cause of hypercalcaemia. However, the frequency of this event may increase as high-dose unactivated vitamin D preparations become available. Prolonged vitamin D toxicity can cause reversible hypercalcaemia and partially reversible renal impairment. Parathyroid hormone may not be suppressed with unactivated vitamin D toxicity, especially if renal disease coexists. © 2013 Royal Australasian College of Physicians.

Original language	English
Pages (from-to)	1148-1150
Journal	Internal Medicine Journal
Volume	43
Issue number	10
DOIs	https://doi.org/10.1111/imj.12269
Publication status	Published - 2013

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10.1111/imj.12269

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AB - Vitamin D toxicity from unactivated vitamin D (calciferol) therapy is currently a rare cause of hypercalcaemia. However, the frequency of this event may increase as high-dose unactivated vitamin D preparations become available. Prolonged vitamin D toxicity can cause reversible hypercalcaemia and partially reversible renal impairment. Parathyroid hormone may not be suppressed with unactivated vitamin D toxicity, especially if renal disease coexists. © 2013 Royal Australasian College of Physicians.

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Prolonged vitamin D intoxication: Presentation, pathogenesis and progress

Abstract

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