Immunogenetic traits of a heritable CD56+cell population dysregulated in Multiple sclerosis: clinical utility and implications for pathogenesis

D. Booth; F. McKay; P. Gatt; N. Fewings; G. Parnell; S. Schibeci; M. Barnett; M. Slee; A. Kermode; J. McCauley; G. Stewart; S. Vucic

Immunogenetic traits of a heritable CD56+cell population dysregulated in Multiple sclerosis: clinical utility and implications for pathogenesis

D. Booth, F. McKay, P. Gatt, N. Fewings, G. Parnell, S. Schibeci, M. Barnett, M. Slee, A. Kermode, J. McCauley, G. Stewart, S. Vucic

Centre for Neuromuscular and Neurological Disorders (Perron Institute)

Research output: Contribution to journal › Abstract/Meeting Abstract › peer-review

Original language	English
Pages (from-to)	286
Journal	European Journal of Immunology
Volume	46
Issue number	S1
Publication status	Published - 2016

Cite this

@article{26f99448729a4bc79ba17823bdd3fa98,

title = "Immunogenetic traits of a heritable CD56+cell population dysregulated in Multiple sclerosis: clinical utility and implications for pathogenesis",

author = "D. Booth and F. McKay and P. Gatt and N. Fewings and G. Parnell and S. Schibeci and M. Barnett and M. Slee and A. Kermode and J. McCauley and G. Stewart and S. Vucic",

year = "2016",

language = "English",

volume = "46",

pages = "286",

journal = "European Journal Immunology",

issn = "0014-2980",

publisher = "Wiley-Blackwell",

number = "S1",

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TY - JOUR

T1 - Immunogenetic traits of a heritable CD56+cell population dysregulated in Multiple sclerosis: clinical utility and implications for pathogenesis

AU - Booth, D.

AU - McKay, F.

AU - Gatt, P.

AU - Fewings, N.

AU - Parnell, G.

AU - Schibeci, S.

AU - Barnett, M.

AU - Slee, M.

AU - Kermode, A.

AU - McCauley, J.

AU - Stewart, G.

AU - Vucic, S.

PY - 2016

Y1 - 2016

UR - https://publons.com/publon/4889950/

M3 - Abstract/Meeting Abstract

VL - 46

SP - 286

JO - European Journal Immunology

JF - European Journal Immunology

SN - 0014-2980

IS - S1

ER -

Immunogenetic traits of a heritable CD56+cell population dysregulated in Multiple sclerosis: clinical utility and implications for pathogenesis

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