Abstract
Hannen et al. report that cutaneous glucocorticoidogenesis and expression of glucocorticoid receptors are inhibited in psoriatic skin. These findings substantiate the previous concept that deficient feedback of local proopiomelanocortin and glucocorticoids on cutaneous immunity contributes to inflammatory and autoimmune dermatoses. Restoration of efficient endogenous glucocorticoid signaling represents a realistic goal in treating psoriasis.
Original language | English |
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Pages (from-to) | 1609-1611 |
Number of pages | 3 |
Journal | Journal of Investigative Dermatology |
Volume | 137 |
Issue number | 8 |
DOIs |
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Publication status | Published - 1 Aug 2017 |