Caffeine with Links to NAFLD and Accelerated Brain Aging

Ian James Martins

Research output: Chapter in Book/Conference paperChapterpeer-review

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Abstract

Nutritional diets are essential to prevent nonalcoholic fatty liver disease (NAFLD) in the global obesity and diabetes epidemic. The ingestion of palmitic acid-rich diets induces NAFLD in animal and human studies. The beneficial properties of olive oil (oleic acid) may be superseded by ingestion of palmitic acid-rich diets. Hepatic caffeine metabolism is regulated by palmitic and oleic acid with effects of these fats on amyloid beta metabolism. Healthy fats such as olive oil may facilitate rapid amyloid beta clearance in the periphery to maintain drug therapy in diabetes and various neurological diseases. Repression of the anti-aging gene sirtuin 1 (Sirt 1) prevents the beneficial properties of olive oil. Brain disorders induce NAFLD and supersede caffeine’s therapeutic effects in the prevention of NAFLD. Delayed hepatic caffeine metabolism in NAFLD and increased
caffeine transport to the brain with aging-induced mitophagy in neurons with induction of type 3 diabetes and neurodegenerative disease.
Original languageEnglish
Title of host publicationNon-Alcoholic Fatty Liver Disease
Subtitle of host publicationMolecular Bases, Prevention and Treatment
EditorsRodrigo Valenzuela Baez
Place of PublicationUnited Kingdom
PublisherIn-Tech
Chapter8
Pages155-179
Number of pages25
ISBN (Electronic)9789535155799
ISBN (Print)9789535139232, 9789535139249
DOIs
Publication statusPublished - 21 Mar 2018

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